Published Paper Details:

A group of abnormalities known as Potter syndrome and Potter phenotype are connected to an unborn child's lack of amniotic fluid and renal failure. Oligohydramnios, or having insufficient amniotic fluid during pregnancy, is different from anhydramnios, which has no amniotic fluid. A developing foetus is supported, padded, and safeguarded by amniotic fluid. It is possible for the foetus to develop physical characteristics like unusual facial traits or skeletal defects when there is insufficient amniotic fluid. The lungs are also underdeveloped (pulmonary hypoplasia) when oligo-anhydramnios is prevalent early in pregnancy, which can cause serious breathing problems. This illness is typically brought on by the loss of both kidneys (bilateral renal agenesis). This is referred to as the "classic Potter syndrome" at times. Polycystic kidney disease, dysplastic or hypoplastic kidneys, and obstructive uropathy, which prevents urine from leaving the bladder and accumulates in the kidneys, can also lead to Potter syndrome. There could occasionally be amniotic fluid leaks later in the pregnancy, but this won't cause Potter syndrome. Due mostly to pulmonary hypoplasia, Potter syndrome is an extremely dangerous disorder that frequently results in infant death at or soon after birth.